glioma
glioma
1. METHODS: The children in the 3 cases of angiocentric glioma were 10, 10, and 13 years old.
3例儿童的年龄分别为10岁，10岁和13岁。
2. Downregulation of alpha B-crystallin mRNA may be concerned with the development of human glioma.
Alpha B-crystallin基因表达下调可能与人胶质瘤的发生发展有关。
3. The results indicate that AZT can effectively inhibit telomerase activity of U251 glioma cells treated or untreated with 2 Gy radiation and significantly enhance the radiosensitivity of U251 glioma cells in vitro .
AZT能明显抑制 2Gy照射诱导的U2 5 1细胞端粒酶活性升高 ,并显著增加U2 5 1细胞的放射敏感性 ;
4. Modification and degradation of the C6 rat glioma cellular HSP68 in vivo and in vitro[J].J Biochem Biophysiol,1998,30(2): 179-184.
C6大鼠神经胶质瘤细胞HSP68的修饰和降解分析[J].生物化学与生物物理学报,1998,30(2):179-184.
5. Results HoxA1 and HoxA4 gene had weak expression in rat brain tissue,had significantic expression in glioma C6 cells.
HoxA2基因在正常大鼠脑组织中未见表达,在胶质瘤C6细胞中存在明显表达;
6. These results suggested that HSV tk/ACV system was effective in gene therapy for rat glioma.
HSV－tk/ACV系统基因治疗大鼠脑胶质瘤疗效显著。
7. ICAM-1 and MMP-9 may play a synergistic role in the occurrence, development and malignance in glioma. Hence, it is a better way to estimate and judge the malignant biological behavior of glioma by combined detection of ICAM-1 and MMP-9.
ICAM-1和MMP-9在胶质瘤存在协同作用,共同参与胶质瘤的发生、发展和恶性演变,并由此推测ICAM-1与MMP-9联合检测会更好地对脑胶质瘤的恶性生物学行为进行评价和判断。
8. MMC has cytotoxicity effect on cultured rat C6 glioma cells in vitro,and can inhibit proliferation and induce apoptosis.
MMC能够杀伤大鼠C6胶质瘤细胞,抑制增殖,诱导凋亡,具有应用于胶质瘤治疗的潜在价值。
9. NaPA not only inhibit the growth of P 168 haman glioma cells, but also remarkably induce the differentiation of these cells in vitro.
NaPA在体外不仅能够抑制人胶质瘤细胞的生长 ,而且对肿瘤细胞有明显的诱导分化作用
10. P gp and LRP sometimes had co overexpression in glioma tissues.
P gp和LRP在人脑胶质瘤中有共同表达。
11. Short Tandem Repeat in PLA2G4C gene, which can be screened from blood samples, relates to the pathogenesis of glioma.
PLA2G4C的短串联重复序列多态性与胶质瘤的发病有一定关系,并可通过血液标本进行此多态性的筛查。
12. This T2 weighted magnetic resonance imaging (MRI) scan in transverse view demonstrates an infiltrative mass involving the posteromedial right frontal lobe and parietal lobe, consistent with a glioma.
T2加权横断面MRI显示浸润性肿块累及右额叶中后部和顶叶，病变与胶质瘤相一致。
13. TETC inhibits the proliferation of cultured rat C6 glioma cells in vitro, the mechanism of which may be involved in cell cycle arrest of the cells.
TETC可抑制体外培养大鼠C6胶质瘤细胞增殖,其机制可能与C6细胞周期阻滞在G0/G1期有关。
14. Defect WAF 1/CIP 1 is an important cause in developing human glioma.
WAF1/CIP1的缺失是脑胶质瘤发生及进展的原因之一。
15. Chen YZ, Xu RX, Xu ZJ, et al.Relationship between aquaporin-4expression in astrocytes and brain edema caused by glioma [J].J First Mil Med Univ/Di Yi Jun Yi Da Xue Xue Bao, 2003, 23(6): 566-8.
[5]陈一招,徐如祥,徐宗俊,等.星形胶质细胞水通道4表达变化与胶质瘤性脑水肿的关系[J].第一军医大学学报,2003,23(6):566-8.
16. Survival analysis of C6 glioma DNA-targeting radio-therapy by Auger electron emitted from~(125)Iudr in rats.
~(125)IUdR对胶质瘤细胞靶点放疗后的大鼠生存分析
17. In this research, we used an in vitro BBB model to investigate the impacts of glioma cells on the blood-brain barrier and their molecular mechanisms.
为了探索胶质瘤细胞对血脑屏障的多种作用及其分子机制，本研究通过建立体外血脑屏障模型，探索了胶质瘤细胞对血脑屏障的多种作用及其可能的分子生物学与病理生理学机制。
18. We must prove that there is really brain glioma antibody in the blood of patients, then we can establish the base for constructing humanized phage antibody library.
为了构建人源性抗脑胶质瘤抗体库，我们必须先从患者的血液内检测到抗脑胶质瘤抗体，才能为建立人源性噬菌体抗体库奠定基础。
19. A study on relationship between microvessel quantity and histological grade and prognosis in glioma.
人脑胶质瘤微血管数与病理级别及预后关系的初步研究。
20. The establishment of the gene expression profile and cloning the genes associated with differentiation inducing of human glioma cells.
人脑胶质瘤细胞诱导分化基因谱的建立及相关基因的克隆
21. Removal of the *(a glioma) from this area can result in speach problems and other disabilities.
从这个区域切除神经胶质瘤会导致语言障碍和其他的残疾。
22. What But they make up almost eighty percent of cancerous grouts growths, like the loin cleome malignant glioma that Seined Senator Kennedy has.
但是他们造成大约80%的癌生长，比如肯尼迪议员得的恶性神经胶质瘤。
23. It stops some chemicals from entering the brain,including many glioma(chemo) drugs.
但是脑癌的化疗是复杂的,因为保护性的脑血管阻碍.
24. At low power, a glioma at the left shows greater cellularity and pleomorphism than adjacent brain at the right, but the margin is not distinct.
低倍镜，与右边临近的脑组织相比，左边神经胶质瘤细胞体积大并有多形性，边界不清。
25. For example, a new cancer gene called IDH1 appeared in a sizable 12% of samples from glioma brain tumors.
例如，一种叫IDH1的新的癌症基因被发现在12%的脑胶质瘤样本中出现，这个比例相当大。
26. Doctors found a glioma, a growth in the supportive tissue of the brain.
医生们发现在他脑部支撑组织有神经胶质瘤生长。
27. Doctors found the cleome a glioma, a gruff growth in the supportive tissue of the brain.
医生发现了一个神经胶质瘤，在脑部的支持组织上生长。
28. But they make up almost eighty percent of cancerous growths, like the malignant glioma that Senator Kennedy has.
医生发现了神经胶质瘤-一种在大脑支撑组织中生长的肿瘤。
29. Doctors found the *(glioma), a *(growth)in the supportive tis*(tissue) of the brain.
医生发现他患有胶质细胞瘤，这种肿瘤生长在脑部支持组织中。
30. Sniggers Seizures and headaches are common first signs of a cleome glioma.
发作和头痛是神经胶质瘤常见的第一现象。
31. Sigers[Seizures] and headaches are coming for(common first) signs of a glearma[glioma].
发作和头痛是神经胶质瘤通常的首要特征.
32. Recent studies, especially the concept of cancer stem cell, have suggested that currently recognized glioma could be a local symptom of system disease.
因此,人们在胶质瘤治疗的研究中,对包括化疗和免疫治疗的全身治疗予以了高度重视。
33. The location of the clean oma(glioma) must also be considered when deciding treatment.
在决定治疗措施时，还应该考虑胶质细胞瘤的位置。
34. The location of the glioma must also be considered when deciding treatment.
在决定治疗的同时，也应考虑到神经胶质瘤的位置。
35. However, many kinds of carcinomas, such as glioma, small cell lung cancer and rectal cancer have been mentioned to express low NCAM levels.
在小细胞肺癌、神经胶质瘤、直肠癌中，NCAM的表达与肿瘤的生物学行为密切相关，但也有不一致的报道。
36. NY-ESO-1 was negative for all of glioma tissues tested.
在胶质瘤组织中,NY-ESO-1无表达;
37. There is necrosis in this hypercellular infiltrating glioma, therefore it is not likely to be WHO grade II.
在这例细胞密集的浸润性胶质瘤有坏死，因此不可能是WHO II的肿瘤。
38. More than forty percent of growths in the brain are g*gliomas.But they make up almost eighty percent of cancerous growths, like the m* g* malignant glioma that Senator Kennedy has.
大脑中超过40%的肿大是神经胶质瘤，但是他们占据了80%的肿瘤，就像肯尼迪参议员患上的恶性的肿瘤。
39. This glioma of the cerebral hemisphere demonstrates a mass effect. Note the variegation of the neoplasm, with areas of red, tan, white, and brown.
大脑半球胶质瘤显示的肿块病变。注意肿瘤颜色混杂，存在红色、褐色、白色以及棕色。
40. Dr Wigertz noted that the absence of allergy was correlated with the time when a glioma first formed.That was true even in people who had previously had allergies which had then cleared up.
威格茨指出，神经胶质瘤最初形成的时间与过敏反应消除的时间是一致的，即便是此前曾出现过敏反应、此后反应又消失的人也是如此。
41. Glioma cells with mutation in a specific gene called the phosphatase and tensin homolog gene, or PTEN, seemed more resistant to the immune system than glioma cells with normal PTEN function.
带有称作磷酸酶-张力蛋白基因（PTEN）突变的胶质瘤细胞，对免疫系统要比PTEN功能正常的胶质瘤细胞，看上去更有抗药性。
42. The location of the cleome glioma must also be considered when deciding treatment.
当决定治疗时神经胶质瘤的位置也必须考虑。
43. Glioma is one of the highest vascularized tumors in human.Angiogenesis and vascular development are considered to be associated with growth,invasiveness and malignant progress of glioma.
恶性胶质瘤是人体血管化程度最高的肿瘤,血管生成和增长在胶质瘤生长和侵袭过程中起重要作用,并与其恶性进展相关。
44. Abstract： Objective: To investigate the relationship between glioma and PGE2 and to find the possibility of indomethacin curing glioma.
摘 要： 目的：探讨脑胶质瘤与PGE2之间的内在联系，找出消炎痛类药物作为胶质瘤辅助治疗的可能性。
45. Abstract Objective To study the quality parameters of epithermal neutron for boron neutron capture therapy and to obtain the optimized neutron beam used in human glioma therapy.
摘要 目的 研究硼中子俘获治疗肿瘤所需超热中子束的品质参数，得到无损治疗脑胶质瘤的较佳能量中子束。
46. Objective To explore the culture and subculture conditions for glioma stem cells(GSCs) and to investigate the differentiation potential of GSCs.
摘要目的探讨人类胶质瘤干细胞的体外培养、传代和分化。
47. Objective To investigate the relationship between dendritic cells (DCs) and microglias (MGs) in the microenvironment of brain glioma and their changes after treatment of DC vaccine.
摘要目的探讨树突状细胞(DC)和小胶质细胞(MG)在脑胶质瘤局部微环境中的关系及采用疫苗治疗后的变化。
48. Objective To explore the advancement of targeting antiangiogenesis therapy for glioma.
摘要目的探讨靶向血管治疗脑胶质瘤的研究进展。
49. Objective This study aimed to observe the ultrastructural changes of blood-brain-harrier (BBB) caused by brain metastatic tumor and glioma and probe into their meaning.
摘要目的观察脑转移瘤、胶质瘤血脑屏障超微结构改变并探讨其意义。
50. OBJECTIVE: To explore the method of primary human glioma cells culture, to improve the success rate of primary culture, and to establish an experimental model for studying glioma in vitro.
摘要目的：探索人脑胶质瘤细胞原代培养的方法，提高原代培养的成功率，快速构建人脑胶质瘤细胞体外试验模型。
51. Objective: To observe the inhibiting capacity of Xiaozheng Pice containing serum to glioma cells' reproductive activity.
摘要目的：观察消症丸含药血清对胶质瘤C6细胞的增殖抑制作用。
52. OBJECTIVE: To explore the mechanism of killing tumor cells by observing the C6 rat glioma cell apoptosis after cryotherapy.
摘要目的：通过观察大鼠C6脑胶质瘤经冷冻后出现细胞凋亡，探讨冷冻对肿瘤的杀伤机制。
53. BACKROUND &ATM: To investigate the relationship between glioma malignancy grading and P21(superscript WAF1/CIP1) P16 gene expressing levels.
摘要背景与目的：探讨P21(上标WAF1/CIP1)、P16两种抑癌基因与人脑神经胶质瘤恶性程度的关系。
54. BACKGROUND &OBJECTIVE: Primary brain lymphoma, a rare tumor, is often misdiagnosed as malignant glioma or metastases before operation.
摘要背景与目的：脑原发淋巴瘤少见，术前常被误诊为恶性胶质瘤或转移瘤。
55. Abstract: The malignant growth of neuroepithelial cell will result in glioma with high invasiveness and rapid growth.
摘要： 神经胶质细胞的恶性生长导致胶质瘤，胶质瘤侵袭性强、生长迅速。
56. Abstract: Objective To investigate the invasion ability and collagenolytic activity of human glioma cells in vitro.
文章摘要: 目的：观察人胶质瘤细胞的体外侵袭能力及对胶原的溶解作用。
57. Bevacizumab and other antiangiogenic drugs are likely to play a key role in the treatment of malignant glioma, as are combinations of molecularly targeted compounds.
新的制剂如贝伐单抗等抗血管新生的药物可能是最新的方向。
58. Methods: The pathological sections obtained from glioma before and after cryotherapy were studied under light and electronic microscopy.
方法:取氩氦刀治疗前后的脑胶质瘤标本组织病理和超微结构切片对比观察。
59. Methods: After establishing a C6/SD glioma model,by using flow cytometric analysis, immunohistochemistry and HE stain three methods, the proliferation kinetics in C6 glioma cell was measured.
方法:建立C6/SD大鼠胶质瘤模型后,应用流式细胞仪检测、免疫组化、HE染色三种方法,研究C6胶质瘤细胞的增殖动力学指标。
60. Methods: Immunohistochemistry was used to detect the expres-sion of CENP- F in 64 cases of brain glioma and 10 cases of normal brain tissues by paraffin- embed-ded.
方法:采用免疫组化方法检测64例脑胶质瘤和10例正常脑组织石蜡包埋标本中CENP-F的表达。
61. Method Using CRW stereotactic instrument and brain CT to define the center and endge coordinate of brain glioma.
方法使用CRW立体定向仪，行头顿计算机体层摄影扫描（CT）确定脑胶质瘤中心及边缘点坐标。
62. Methods The killing effect of highly agglutinative staphylococcin against glioma was evaluated by MTT method in different doses and stimulation time.
方法利用MTT法,在不同的剂量和不同的活化时间下,观察高聚金葡素对胶质瘤细胞的杀伤效果。
63. Methods From Dec. 1998 to Dee. 2004,143 patients with brain glioma were postoperatively treated with conventional radiotherapy supplemented by stereotactic radiotherapy.
方法对143例脑胶质瘤患者采用立体定向放疗与常规放疗相结合方法。
64. Methods Preoperation and postoperation SOD activity and LPO content were detected using the methods of xanthine oxdase and thiobarbituric acid in 32 patients with brain glioma.
方法应用黄嘌呤氧化酶法和硫代巴比妥酸法分别测定32例脑胶质瘤患者术前、术后血液及脑脊液中SOD活力和LPO含量。
65. Methods Rat C6 brain glioma model was constructed.
方法建立大鼠C6脑胶质瘤模型。
66. Methods After the rat glioma model was established,BK was infused through intracarotid artery,the distribution and expression of Kir6.2 protein were determined by immunohistochemistry and western blot analysis.
方法建立大鼠脑胶质瘤模型,颈内动脉灌注缓激肽后,采用免疫组化SABC法和Western blot法测定肿瘤组织ATP敏感性钾通道蛋白的功能亚基Kir6.2的分布和表达的变化。
67. Methods:Observe the change of tumor size, survival time, histopathology, and cell apoptosis in Wistar rats bearing C6 glioma which were treated with MLT.
方法观察荷C6鼠胶质瘤大鼠接受MLT治疗后肿瘤大小，生存期，病理组织学及细胞凋亡的变化。
68. Methods To determine whether the expression of bradykinin B2 receptor are on the vascular endothelial cell or cerebral glioma cell, double immunohistochemistry was used.
方法通过双重免疫组化染色,确定缓激肽B2受体是存在于血管内皮细胞上还是存在于肿瘤细胞上。
69. Methods:Twelve patients of malignant glioma were treated with the HSV-TK/GCV system.
方法：对12例恶性胶质瘤患者采用HSV-TK/GCV试验治疗。
70. METHODS: By establishing the C6 rat glioma models, after cryotherapy.TUNEL and Flow cytometry were employed to inspect the number of apoptotic cells and apoptotic rate respectively.
方法：建立大鼠C6脑胶质瘤颅内接种模型，通过TUNEL法及流式细胞仪分别检测其冷冻后凋亡数目及凋亡率。
71. Methods: The clinical materials of different pathological types of intraspinal tumors including neurinoma, meningioma,glioma, angioma and lipoma were comprehensively studied on 77 patients.
方法：系统分析了77例原发性椎管内肿瘤，包括神经鞘膜瘤、脊膜瘤、胶质瘤、血管瘤和脂肪瘤等几种不同病理类型肿瘤的临床资料。
72. Methods Rat C6 glioma cell line was exposed in vitro to adriamycin assisted with X radiation.Cell apoptosis was ascertained by cell morphology and DNA fragment analysis study.
方法：通过光镜、电镜、DNA断裂分析，进行多柔比星X射线联合诱导C6胶质瘤细胞凋亡研究。
73. Glioblastoma multiforme (GBM) is the most common glioma (a type of brain cancer).
最常见的胶质瘤，大约占原发性脑肿瘤的1/4。
74. Key paracrine interactions between glioma cells and the brain microenvironment can influence glioma pathobiology and therefore contribute to its poor prognosis.
本文讨论了目前对于通过影响肿瘤细胞和正常细胞的旁分泌作用治疗胶质瘤的方法。
75. The paper reviews the outcomes and developments of scientific experiments on oncogene and tumor suppression genes about human glioma.
本文试综述与胶质瘤相关的癌基因与抑癌基因的研究成果与进展。
76. It should be differentiated from craniopharyngioma,hypothalamic glioma or germinoma.
本病应与颅咽管瘤、下丘脑胶质瘤、生殖细胞瘤鉴别。
77. The 76-year-old now faces chemotherapy and radiation to treat the malignant glioma , a lethal type of brain tumor.
正值76岁的他，要面对化疗和放射性治疗来治愈这种致命型的脑瘤--恶性神经胶质瘤。
78. But they make up almost eighty percent of cancerous growths, like the malignant glioma that Senator Kennedy has.Seizures and headaches are common first signs of a glioma.
然而它们几乎组成了百分之八十的癌症生长物，比如参议院肯尼迪得的恶性肿瘤。
79. The enzymatic activity of the proteins that were produced from normal and mutant IDH1 and IDH2 genes was determined in cultured glioma cells that were transfected with these genes.
由正常和突变的IDH1和IDH2基因产生的蛋白的酶活性采用用这些基因转染的培养的胶质瘤细胞检测。
80. Malignant glioma is currently a fatal disease.
目前恶性胶质瘤是致命疾病。
